You said this and you've not shown any proof of it yet... I also said that if you're changing the type of virus/bacteria to something that is not an upper respiratory infection, then that's not the same thing as what I was claiming, then you link to something about noroviruses.
Again, you've shown nothing to prove your claim like you always do. You have no idea for most viruses/bacteria what the ACTUAL infectious dose is because challenge studies are rarely done. You have no idea how much actual live virus is in the air/surfaces per shedding event (breathing/coughing/sneezing/touching) or how much a non-infected person gets per interaction (breathing in/touching/etc) that actually make it inside the body in some form.
We already have, in this thread, a link to a metastudy encompassing numerous studies about infectious dose and various transmission vectors-- aerosol and fomite both. Then I provided 2 more-- one of which concerns norovirus, yes, but the other concerns rhinoviruses. When you say nothing has been provided, you aren't being truthful.
If you mean we haven't got a study looking at how much active viral material gets into a passerby in a naturalistic, real setting, that's obviously not practical. But we damn well have enough if we know roughly how much is in a sneeze, and how much is needed to create an infection in an experimental setting. We can derive conclusions. We can extrapolate. Its the best we can do within reason, and its how epidemiology and medical science has always been done.
TL;DR: You're essentially arguing that since we don't have a study looking at exactly the same situation as a naturalistic encounter (even though that's not possible), therefore we should just assume there's no risk, even though other methods of analysis show quite a lot of risky material.